Symptoms The disease is a bacterial disease that is accompanied by wheat nematodes. Wheat occurs after heading. Infected strains were curled in the heart, yellow gums and bacteria overflowed between the leaves and sheaths. Fresh leaves are obstructed when they are drawn from the pus tube containing the above bacteria, and often have bacterial secretions. The diseased wheat ear is thin or unable to be extracted, and the glaze is often stained with a yellow glial substance. After drying, the pus overflows and turns into a white film on the ear or upper leaf, so that the ear and leaf are firm. When the humidity is high, the amount of pus overflowing or flowing drops. When the wheat matures, the yellow gum condenses into colloidal pellets.
Pathogen Clavicacter tritici (Carlson and Vidaver) Davis et al. call it Corynebacterium anatipestifer, belonging to the genus Corynebacterium, is a short wedge-shaped bacillus with blunt ends, size 1.30.5-0.7 (μm), usually single or end-to-end arrangement. Gram-positive, aerobic, non-spore-producing, bacterial stick-like, no spherical or rod-shaped polymorphisms, and no sudden breaks in the division. The cell wall contains diaminobutyric acid; G+C is 51% to 59%. The bacterium was originally named Corynebacterium tritici (ex. Hutchinson). Cahson and Vodaver were included in the newly established Corynebacterium genus.
Transmission routes and disease conditions The disease mainly depends on wheat nematodes as mediators to invade wheat, and the rapid expansion of bacteria after intruding results in head panicle disease. When the nematode develops rapidly, the diseased ear becomes worm-eaten or partially worm-eaten, and part of it is honey-ear. The head worms in the head stalk strains are all pathogenic bacteria of the head blast disease. Can survive in the gall of about two and a half years.
Prevention and control methods focus on the prevention and control of wheat nematodiasis, wheat nematode disease after the prevention, that there is no occurrence of this disease.
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